Finally, when all of the 64 blood cell samples studied are compared with respect to two factors -- (i) the mean SD values for TP53, CEN17 and AML1; and (ii) the mean aneuploidy value of chromosomes 17 and 21 -- it is clearly seen that the cancer-related aberrant SD value (an epigenetic marker) is set back to the normal following transplantation, while the cancer-associated increase in aneuploidy level (a genetic marker) is not readjusted to the background level (Figure 4). The gene discussed is RUNX1; the disease is cancer.