GLO1 and congenital nonspherocytic hemolytic anemia: Thus, our findings of a posttranslational modification of Glo1 by GSH could be important for the many diseases, in which altered Glo1 activity was observed and, consequently, obviously might need to be regulated in response to the redox state of tissues beyond protein expression, such as tumor cell proliferation [6], diabetes [52], congenital nonspherocytic hemolytic anemia [53], and numerous others.