While this work was in preparation, three new miR-210 targets were validated, i.e. HOXA1, HOXA9 and FGFRL1, and the analysis of tumour xenografts derived from cancer cell lines overexpressing miR-210 suggested a potential involvement of miR-210 in tumour growth initiation [40]. The gene discussed is FGFRL1; the disease is neoplasm.