It is known that in cases of ITP, antiplatelet antibodies are produced, which may have specificity for either GPIIb/IIIa or Ib/IX complexes.[4] GT is a rare disorder and hence this was a unique opportunity to investigate these patients, both multitransfused and ones who were never transfused with blood or platelet concentrates. The gene discussed is ITGA2B; the disease is autoimmune thrombocytopenic purpura.