Measurement of immunoreactive TGF-β1 in bronchoalveolar lavage fluid revealed that the basal TGF-β1 level is significantly elevated in atopic asthma patients and it increases further in response to allergen exposure.[6] Th2-dominated inflammation is well illustrated to be the cornerstone of the disorder [7], with airway remodeling believed to be an adverse consequence of the inflammatory response [8]. The gene discussed is TGFB1; the disease is atopic asthma.