Also, it is becoming increasingly accepted that tumor cells may not necessarily undergo a complete transition, but rather seem to adopt different "quasi-mesenchymal" states which, in addition to the transient nature of EMT and the contribution of other E-cadherin repressors, may explain the lack of correlation between SNAI1 expression and the mesenchymal phenotype, including E-cadherin loss. The gene discussed is SNAI1; the disease is neoplasm.