Representative data from one time point of infection shows that there was complete inhibition of SDF-1, SCF, TGF-β and TARC with 60–70% inhibition of GM-CSF, GRO, GRO-α, ILs (−2,−3, −4,−5, and −10), MDC, MIG, MIP-1Δ, RANTES, IGF-1, angiogenin, oncostatin-M, and TPO and 30–40% inhibition of VEGF, PDGF-BB, IL-7, IL-1β, and IL-8. The gene discussed is CXCL8; the disease is infection.