This function would not be selected for in tumour development if the action of Myc was redundant with that of EBER1 (the latter activity already being present in the EBER1 expressing tissues) and as such the data indicate that Myc and EBER1 action are not redundant in tumourigenesis and potentially could cooperate in ways not assessed by the tumour latency assay described. Here, MYC is linked to neoplasm.