As an inflammatory process, acute pancreatitis results in an excessive leukocyte activation and increased migration of neutrophils to the inflamed area with a consequent release of proinflammatory mediators including interleukins (IL-1b, IL-6, IL-8, IL-10, IL-18) and Tumor Necrosis Factor-alpha (TNF-α) [4–11]. Here, TNF is linked to acute pancreatitis.