Various physiopathological mechanisms have been incriminated for the different behaviors of diabetics, such as: the exaggerate vascular inflammatory reaction (also shown by a higher growth of C-reactive protein in patients with diabetes), the increase of platelet aggregation and the stimulation of IIbIIIa glycoprotein and CD40 ligand molecule – factors that contribute to thrombus formation [4,5]. This evidence concerns the gene CD40LG and diabetes mellitus.