IFNL2 and severe acute respiratory syndrome: Taken together, these results suggest that while the expressions of IFN-λ2 and, especially of IFN-β proteins, were profoundly inhibited, SARS-CoV infection did not impose a generalized suppression of host posttranscriptional machinery in 2B4 cells, an observation consistent with that reported for SARS-CoV nsp-1 protein [12], [64].