IL1A and severe acute respiratory syndrome: The likelihood of SARS being an immune-mediated disease was further supported by reports, within the circulation and the lungs of patients affected by SARS, of highly elevated expressions of various inflammatory mediators, including interleukin (IL)-1, -6, -8; CXCL-10/Interferon-inducible Protein (IP)-10; CCL2/Monocyte Chemoattractant Protein (MCP)-1; CCL5/Regulated on Activation, Normal T Expressed and Secreted (RANTES); and CXCL9/Monokine Induced by interferon-Gamma (MIG) [9], [10], [11], [12], [13], [14].