More specifically, we were particularly interested in comparing virally induced genes that are either encoding for or relevant to the expression of IFN and other inflammatory cytokines as SARS pathogenesis has been proposed to stem from the combination of barely detectable, if any, IFN and exacerbated cytokine responses in patients severely affected by SARS-CoV infection [14], [47]. This evidence concerns the gene IFNA1 and severe acute respiratory syndrome.