The BCR/ABL1 fusion, housed in the Philadelphia chromosome and resulting from t(9;22)(q34;q11) or variants, is the hallmark of chronic myeloid leukemia (CML) and the most frequent recurrent genetic aberration in B lymphoid leukemia in adults [1,2]. Here, ABL1 is linked to chronic myelogenous leukemia, BCR-ABL1 positive.