The present study adds to an accumulating body of evidence from gene-array studies that genes such as CLCA1 [35], [36], SerpineB2 [26], [36], [37], MUC5AC [35], [37], AGR2 [35], CPA3 [36], [37], and tryptase [36], [37] are overexpressed in asthma, and suggests that these genes are more transcriptionally active in the EIB phenotype. This evidence concerns the gene CPA3 and asthma.