Its role in phagocytosis of blood borne antigens, synthesis of antibodies and opsonins such as tuftsin and properdin are now well established.[19] The emphasis in the present day is on splenic conservation procedures due to the supposedly high incidence of post-splenectomy sepsis (4.4% in children under 16 years and .9% for adults).[20] Amongst these procedures, there are reports of partial splenectomy in various disorders.[1, 21, 22] Though these series demonstrate satisfactory results, partial splenectomy for hematological disorders leaves a number of issues unresolved. The gene discussed is CFP; the disease is Sepsis.