Although transactivation of mir-30c-1 and mir-15b-16-2 genes was observed in C. parvum-infected cells and potential NF-κB binding sites were identified in their promoter elements, inhibition of p65 activation failed to inhibit transactivation of either mir-30c-1 or mir-15b-16-2 in H69 cells following C. parvum-infection. The gene discussed is NFKB1; the disease is infection.