Although much emphasis has been rightly placed on the effects of DGC disruption on skeletal and cardiac muscle, many DMD patients have substantial neurological problems, including learning difficulties [12,13] (30% of DMD patients have an IQ<70, and in 6%, IQ<50; this correlates strongly with mutation site, implicating short dystrophin isoforms in cognitive function [13]), congenital stationary night-blindness [51], defective colour vision [52] and a suggestion of personality disorders [53-55]. The gene discussed is DMD; the disease is congenital stationary night blindness.