It is known that a sustained, constitutive activation of NFκB contributes to malignant progression and therapeutic resistance in most of the major forms of human cancer, such as human lymphomas [31], carcinomas of the breast [32], prostate [33], lung [34], colon [22], pancreas [35], thyroid [21], head and neck [36] and cervix [37]. Here, NFKB1 is linked to breast carcinoma.