TNFRSF1B and myeloid sarcoma: Among the genes upregulated were IL-1 receptor (IL1R), IL-8 receptor type 2 (IL8RB), IL-11 receptor α (IL11RA), IL-17, and p75 tumor necrosis factor–receptor transcripts (TNFR1B), indicating involvement of different pro-inflammatory cytokines in MS pathogenesis.