The Actin>CD2>Gal4 “flip-out” chromosome [44] was used in combination with a UAS-GFP transgene and a transgene carrying a dominant-negative form of dome that lacks the C-terminal tail (UAS-domeΔCYT) [45] to produce GFP-positive/domeΔCYT-expressing clones in the background of an ept tumor. The gene discussed is CD2; the disease is neoplasm.