Treatment of mice with α-IL-17 monoclonal antibody significantly reduced KC expression in response to infection (Fig. 7A) and this corresponded to a significant reduction in neutrophil recruitment to the airways (Fig. 7B), indicating that IL-17 plays a role in inducing chemokine expression and subsequent neutrophil recruitment in response to WT B. pertussis at the peak of infection. This evidence concerns the gene IL17A and infection.