Although a heterogeneous cell population (see before), NCAM+EpCAM− cells highly overexpressed (>five fold) most MM stem/progenitor genes in five separate HFK (Fig. 6a), levels of which were already reduced in the NCAM+EpCAM+ cell fraction (presumably more differentiated), but still higher (Wt1, Sall1) in comparison with the NCAM− cell fraction (Fig. 6a), indicating a hierarchy for enrichment for the renal ‘progenitor’ genes. The gene discussed is SALL1; the disease is Miyoshi myopathy.