NFKB1 and hydrops fetalis: The reciprocal activation of the inflammatory/immune response and the lipogenic/adipogenic pathways emerges as the most prominent signature of the transition from short to long-term HF feeding and underscores the relevance of the antagonistic action of NF-κB and PPARγ regulators in controlling the shift from the stressed, inflamed to the pathological, steatotic hepatic state.