We concentrated on the Th1 and Th2 effector cytokines IFNγ and IL-4, which play important roles in anti-malarial immunity and immunopathology [37]–[40], as well as the Th17 cytokine IL-17, which promotes the recruitment of neutrophils [41] that contribute to the clearance of malaria-infected RBCs as well as malaria pathogenesis [42],[43]. This evidence concerns the gene IFNG and malaria.