However, prenatal exposure of mice to human influenza virus on GD18 alters the expression of genes associated with schizophrenia and autism such as Sema3a, Trfr2 and Vldlr [65] and both GD9 and GD18 exposures cause up-regulation of the transcription factor Foxp2 [65], [66] which has been linked with schizophrenia [67] and autism [68]. Here, VLDLR is linked to autism.