Since expression of PPAR isoforms are commonly dysregulated in human carcinomas [34] and novel therapeutic options are provided by the anti-diabetic drug class of thiazolidindiones, further studies are clearly warranted to identify the role of B- and L-FABP in relation to PPAR isoforms in carcinogenesis and progression of renal cancer. This evidence concerns the gene FABP1 and renal carcinoma.