Furthermore, we found that poorer survival was associated with decreased expression of two immune response-related genes, IFNGR1 and “ras-related C3 botulinum toxin substrate 2” (RAC2) and increased expression of 21 genes related to cell cycle (15/23 genes), DNA metabolism (12/23) and repair (4/23), and cell growth/maintenance (16/23), which is consistent with recent reports showing that those biological functions are essential for tumor progression and patient survival [26]–[29]. The gene discussed is RAC2; the disease is neoplasm.