To determine the involvement of p55 TNFR in the AngII-induced model of AAA formation, LDLR−/− mice either wild-type (p55+/+LDLR−/−) or deficient for p55 TNFR (p55−/−LDLR−/−) were fed a fat-enriched diet and infused with either saline (n = 3/group) or AngII (1000 ng/kg/min; n = 13–14/group) for 28 days. Here, AGT is linked to triple-A syndrome.