CASP3 and infection: Although Casp3−/− DCs exhibited enhanced resistance to cell death induced by L. pneumophila ΔflaA, they were not as resistant to cell death as the Bax−/−Bak−/− DCs (Figure 6B), which likely explains why the Bax−/−Bak−/− DCs were slightly more permissive for replication of L. pneumophila ΔflaA at 12-hours post infection compared to the Casp3−/− DCs.