B3GNT2 and parasitic infectious disease: In addition to strong expression of IFNβ and CCL5 in all cell types, parasite infection induces the expression of genes involved in cell-matrix interactions (osteopontin; SPP1) carbohydrate modification (B4GT5 and B3GNT2), vesicular transport SYTL3 and T-SNARE1 and Ca2+ homeostasis STIM1 and CARKL (Table 1).