First, we used AGS gastric carcinoma cells instead of HeLa cervical cancer cells; second, we generated cells that stably expressed shRNAs instead of transient transfection of siRNAs; and third, while the previous report addressed only scattering (which by itself did not seem robust), we performed both scattering and motility assays to determine the role of c-Met in H. pylori-induced cell scattering and motility. This evidence concerns the gene MET and gastric carcinoma.