SKIL and ovarian carcinoma: The first peak contains eight significantly over-expressed genes, EVI1, MDS1, MYNN, TLOC1, GPR160, PHC3, PRKCI, and SKIL. PRKCI has been shown to be a target of amplification in ovarian cancer that contributes to transformation in cooperation with mutant Ras in addition to contributing to anchorage independent growth [17].