Moesin, a member of the FERM (4.1 protein, ezrin, radixin, moesin) domain-containing family of proteins interacting with cytosolic tails of trans-membrane proteins, can activate the IL-2/IL-2R pathway [48], a system well known to be inefficient in maintaining Treg cells in this model of autoimmunity. Here, MSN is linked to Autoimmunity.