Additionally, the BALB/c mice immunized with anti-B19-VP1u IgG developed thrombocytopenia, prolongation of aPTT, and autoantibody against β2GPI and PhL and suggested the association among anti-B19-VP1u IgG and production of anti-β2GPI antibodies, APhL, and APS-like autoimmunity [13]. This evidence concerns the gene APOH and Thrombocytopenia.