The aim of our study was to investigate the role of ING4 in the pathogenesis of pulmonary fibrosis by assessing its expression both in the bleomycin (BLM)-model and in two different types of human pulmonary fibrosis by using tissue microarrays, quantitative reverse transcription -(qRT)-polymerase chain reaction (PCR) and immunohistochemistry. This evidence concerns the gene ING4 and pulmonary fibrosis.