CACNA1A and infection: Finally,we found that HDL stimulated HCVpp entry (Figure 3 and Figure S4)and HCVcc (Figure 4) atsimilar levels for SR-BI cytoplasmic tail mutants as compared to wt SR-BI.Altogether, these results indicated that the C-terminal cytoplasmic tail ofSR-BI modulates the basal HCV entry process, but seems not to influenceHDL-mediated infection-enhancement.