While CD36 expression inSK-Hep1-CLDN1 cells did not induce infection of HCVcc, the SR-BI/CD36 chimeraand the SES mutant were functional, but, for the latter, infection was reducedin comparison to wt SR-BI (Figure4), in line with results obtained with HCVpp infection assays. This evidence concerns the gene CD36 and infection.