CXCL9 and bacterial infectious disease: In PTs but not in AGMs, the peak of viremia (day 10) was associated with enhanced transcription of genes associated with acute inflammation and bacterial infection (e.g., IL-1β, IL-22, S100A8-9) [30], and with the chemotaxis of activated and differentiated Th1 and myeloid cells (e.g., CXCL9/MIG, CXCL11/I-TAC, CCL23/MPIF-1), while transcriptional levels of some genes associated with the chemotaxis of resting T cells and immature dendritic cells (e.g., CCL28/MEC, CCL20/LARC) were reduced (Figure 2C, R1–R2).