In RVFV-infected cells, the NSs establishes general transcriptional suppression at a later stage of infection (after 8 h.p.i.)[25], while NSs also suppresses specific IFN-β mRNA transcription at early stages of infection (about 3 h.p.i.)by maintaining repressor complex including SAP30 on IFN-β promoter [26],[45]. Here, IFNB1 is linked to infection.