Thus in the present study, cell cycle, Wnt, Jak-STAT, and MAPK pathways, playing a role in apoptosis, proliferation, differentiation, and/or cell cycle, were identified as having a role in ovarian cancer; consistent with these results, aberrant signaling of each of these pathways has been proposed as contributing to ovarian carcinogenesis [46–50], again indicating the strength of our approach. The gene discussed is SOAT1; the disease is ovarian carcinoma.