Influenza infection by the intravenous route did not affect proliferative expansion of the CCR7-competent CL-4 CD8+ T cells in the MLN of the CCR7-deficient host (Fig. S8B), suggesting that the absence of the CCR7 receptors does not alter the ability of LNDC to interact with and activate naïve CD8+ T cells, at least under conditions of intravenous virus infection. The gene discussed is CD8A; the disease is influenza.