Exogenous administration of TGF-β1 may interact with the endogenous system [31], upregulate cell adhesion molecule (CAM) expression [32], increase the potency of other neurotrophic factors involved in neurogenesis [33], modulate their action, and affect the signaling of classic neurotrophins [34], and thus enhance stroke-induced neurogenesis. Here, TGFB1 is linked to Stroke.