Excluding lec-11 from the remaining analysis with transgenic animals, we note that the expression of the fat-3 gene under the control of its own promoter was sufficient to rescue the expression of all but one of the 11 infection-response genes tested (Figure S5A), indicating that the requirement for fat-3 in immune function is strongly correlated with the expression of infection-response genes. This evidence concerns the gene FAT3 and infection.