In an experimental model of leukocyte infection, it was recently suggested that BCG (Bacille Calmette Guerin) infection can induce, in a TLR2-dependent fashion, the rapid formation of lipid bodies carrying out part of the eicosanoïd biosynthesis that usually accompanies the infection, thus pointing to an active role for lipid bodies during the course of infection [16]. The gene discussed is TLR2; the disease is infection.