As we have shown a perinuclear aggregation of both PKCε and vimentin under galectin‐1 depletion that is similar to the vesicular aggregation of these two molecules seen by Ivaska et al under inhibition of PKCε activity that, in their study, lead to decreased integrin recycling, we can implicate galectin‐1 in mediating the PKCε–vimentin‐controlled integrin‐β1 recycling pathway in glioma (18). This evidence concerns the gene VIM and glioma.