Heterozygous SLC4A11 mutations have recently been reported in Fuchs endothelial corneal dystrophy (FECD), a late-onset progressive disorder of the corneal endothelium, indicating that carriers parents of affected CHED2 or Harboyan children might be at risk of developing late-onset corneal dystrophy [35]. Here, SLC4A11 is linked to Fuchs' endothelial dystrophy.