It was reported that in animal stroke models, the transplantation of human bone marrow stromal cells, which secrete basic fibroblast growth factor (bFGF) [16] and vascular endothelial growth factor (VEGF) [17], activates the endogenous expression of bFGF, VEGF and VEGFR2, and consequently promotes endogenous angiogenesis, while very few transplanted cells were incorporated into the host circulation [3]. The gene discussed is FGF2; the disease is stroke disorder.