In this manuscript, the authors reported that AILT and other PTCL have rather similar GEP, possibly sharing common oncogenic pathways. In addition, they found that the molecular signature of follicular T helper cells was significantly overexpressed in AILT. Finally, several genes, such as PDGFRA and VEGF, which are deregulated in AILT and represent potential therapeutic targets, were identified. This evidence concerns the gene VEGFA and mature T-cell and NK-cell non-Hodgkin lymphoma.