IL2 and infection: Results showed that several pro-inflammatory mediators were up-regulated following infection [8], many of which had been previously described such as Interferon-γ (IFNγ) [9], [10], [11], [12], Interleukin-2 (IL-2) [9], [10], Interleukin-12 (IL-12) [12], Interleukin-23 (IL-23) [8] and Interleukin-1β (IL-1β) [13], [14].