It is relevant in this regard that cohort studies have found that risk of cardiovascular events, including myocardial infarction, is associated with increased blood levels of inflammatory cytokines such as interleukin-6 (IL-6), tumor necrosis factor-α (TNF-α) and its receptors, adhesion molecules, and acute-phase proteins such as C-reactive protein (CRP) and fibrinogen (Pai et al. 2004). This evidence concerns the gene CRP and myocardial infarction.