However, the mechanism for CAV1 and CAV2 expression in basal-like cancers may stem from maintenance of a basal/myoepithelial phenotype or might be part of a transcriptomic programme of myoepithelial/basal-like differentiation, as these proteins are preferentially expressed in basal/myoepithelial cells of normal breast. This evidence concerns the gene CAV2 and cancer.