PPARγ also interacts with and is activated byERK5 [92, 93] in order to inhibit (in conjunctionwith WNT signaling factors) the proliferation of lung cancer (NSCLC) cells andinflammation in endothelial cells upon flow (shear stress), indicative of aprotective function of ERK5-PPARγ cooperation. Here, PPARG is linked to non-small cell lung carcinoma.